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Ehjcardiovascrec

Estrogen receptor-α gene transfer into bovine aortic endothelial cells induces eNOS gene expression and inhibits cell migration Abstract Objectives: It has been suggested that estrogen may ...

Cardiovascular Research, Original Article, Enqing Tan, Milind V Gurjar, Ram V Sharma, Ramesh C Bhalla

Date : 15/08/1999
Ehjcardiovascrec

Abstract Objective: Vascular endothelial growth factor (VEGF) induces the release of nitric oxide (NO) from endothelial cells. Methods: To further investigate this interaction, we examined the ...

Cardiovascular Research, Original Article, Alicja Jozkowicz, John P Cooke, Ibeth Guevara, Ihor Huk, Philip Funovics, Otmar Pachinger, Franz Weidinger, Jozef Dulak

Date : 01/09/2001
Ehjcardiovascrec

There was no increase in apoptosis detected in eNOS transduced cells after 24 or 72 h. Conclusion: eNOS gene transfer to vascular SMCs inhibits cell proliferation via upregulation of p27 and p21 ...

Cardiovascular Research, Original Article, Jun’ichi Sato, Kamala Nair, Jay Hiddinga, Norman L. Eberhardt, Lorraine A. Fitzpatrick, Zvonimir S. Katusic, Timothy O’Brien

Date : 01/09/2000
Ehjcardiovascrec

Increased activity of arginase may reduce the availability of l-arginine for NO synthase, thus reducing NO production, increasing formation of reactive oxygen species, and leading ultimately to ...

Cardiovascular Research, TOPICAL REVIEW, John Pernow, Christian Jung

Date : 01/06/2013
Ehjcardiovascrec

Following arterial injury, NO has been shown to serve many vasoprotective roles, including inhibition of platelet aggregation and adherence to the site of injury, inhibition of leukocyte adherence, ...

Cardiovascular Research, Review, Melina Kibbe, Timothy Billiar, Edith Tzeng

Date : 15/08/1999
Ehjournal

Methods and results The atherosclerotic plaque model was established in New Zealand rabbits by balloon injury of abdominal arteries and a high cholesterol (1%) diet. Arginase I overexpression ...

European Heart Journal, Wang, Xu-Ping, Zhang, Wei, Liu, Xiao-Qian, Wang, Wen-Ke, Yan, Fei, Dong, Wen-Qian, Zhang, Yun, Zhang, Ming-Xiang

Date : 07/04/2014 Item size : 281375 bytes
Ehjournal

Atherogenic stimuli, such as oxidized lipoproteins, glycation end products, hypertension, and smoking, cause endothelial dysfunction and apoptosis, vascular expression of adhesion molecules, and ...

European Heart Journal, Crea, Filippo, Andreotti, Felicita

Date : 01/10/2005 Item size : 100919 bytes
Ehjournal

Atherogenic stimuli, such as oxidized lipoproteins, glycation end products, hypertension, and smoking, cause endothelial dysfunction and apoptosis, vascular expression of adhesion molecules, and ...

European Heart Journal, Crea, Filippo, Andreotti, Felicita

Date : 01/10/2005
Ehjcardiovascrec

Many cell culture and animal studies suggest that an increased formation of reactive oxygen species (ROS) promotes smooth muscle cell proliferation and matrix formation. Over the years, however, ...

Cardiovascular Research, Brandes, Ralf P.

Date : 01/07/2011
Ehjcardiovascrec

There was no increase in apoptosis detected in eNOS transduced cells after 24 or 72 h. Conclusion: eNOS gene transfer to vascular SMCs inhibits cell proliferation via upregulation of p27 and p21 ...

Cardiovascular Research, Sato, Jun’ichi, Nair, Kamala, Hiddinga, Jay, Eberhardt, Norman L., Fitzpatrick, Lorraine A., Katusic, Zvonimir S., O’Brien, Timothy

Date : 01/09/2000 Item size : 294003 bytes