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The identification of the molecular structure of many ion channels involved in cardiac excitability and their functional correlation with native ionic currents have made it possible to study the ...

Cardiovascular Research, Review, Ralph F Bosch, Stanley Nattel

Date : 01/05/2002 Item size : 503601 bytes

Atrial cellular arrhythmia mechanisms have not been defined in congenital LQTS. [...]we studied atrial cardiomyocyte electrophysiology in mice with an LQTS3 SCN5A inactivation-impairing mutation ...

Cardiovascular Research, Original Article, Marc D. Lemoine, James Elber Duverger, Patrice Naud, Denis Chartier, Xiao Yan Qi, Philippe Comtois, Larissa Fabritz, Paulus Kirchhof, Stanley Nattel

Date : 01/10/2011

Abstract Aims Although atrial tachycardia (AT) remodelling promotes agonist-independent, constitutively active, acetylcholine-regulated K+-current (IK,ACh) that increases susceptibility to atrial ...

Cardiovascular Research, RAPID COMMUNICATION, Niels Voigt, Ange Maguy, Yung-Hsin Yeh, Xiaoyan Qi, Ursula Ravens, Dobromir Dobrev, Stanley Nattel

Date : 01/01/2008

Abstract Background: Loss of rate-dependent action potential (AP) duration (APD) adaptation is a characteristic feature of atrial tachycardia-induced remodeling (ATR). Including ICR alone in the ...

Cardiovascular Research, Original Article, James Kneller, Hui Sun, Normand Leblanc, Stanley Nattel

Date : 01/05/2002