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Ehjcardiovascrec

Abstract Objectives: IKr blockade is ineffective in preventing ventricular fibrillation elicited by the interaction between acute myocardial ischemia and elevated sympathetic ...

Cardiovascular Research, Original Article, Philip B. Adamson, Emilio Vanoli, Stephen S. Hull, Robert D. Foreman, Peter J. Schwartz

Date : 01/10/1998 Item size : 244125 bytes
Ehjcardiovascrec

Ischemic preconditioning (IP), an endogenous protective mechanism rapidly evoked by a brief period of ischemia, is recognized for its protection in limiting myocardial necrosis, but has also shown a ...

Cardiovascular Research, Review, Zhi-Qing Zhao, Jakob Vinten-Johansen

Date : 15/08/2002 Item size : 626546 bytes
Ehjcardiovascrec

[1] in this issue of Cardiovascular Research Delayed rectifier K currents have reduced amplitudes and altered kinetics in myocytes from infarcted canine ventricle forms the coping-stone in a long ...

Cardiovascular Research, Editorial, Marieke W. Veldkamp

Date : 01/10/2000 Item size : 153557 bytes
Ehjcardiovascrec

Abstract Background: We, and others, have previously reported a strong correlation between increased inter-ventricular dispersion of repolarization and the occurrence of fatal arrhythmia in animal ...

Cardiovascular Research, Original Article, Sumeet S Chugh, Stacey Whitesel, Mark Turner, Charles T Roberts, Srinivasa R Nagalla

Date : 01/02/2003 Item size : 273658 bytes
Ehjcardiovascrec

Abstract Objective Congenital loss of function and drug-induced inhibition of the slowly-activating delayed-rectifier K+ current (IKs) cause impaired cardiac repolarization. KeywordsAdrenergic ...

Cardiovascular Research, Original Article, David J. Gallacher, André Van de Water, Henk van der Linde, An N. Hermans, Hua Rong Lu, Rob Towart, Paul G.A. Volders

Date : 01/11/2007 Item size : 253708 bytes
Ehjcardiovascrec

Reperfusion injury is expressed as endothelial and microvascular dysfunction, impaired blood flow, metabolic dysfunction, cellular necrosis, and apoptosis. Postconditioning may also inhibit ...

Cardiovascular Research, Review, Zhi-Qing Zhao, Jakob Vinten-Johansen

Date : 01/05/2006 Item size : 477372 bytes
Ehjcardiovascrec

Abstract Objective: Although studies in vitro have implicated oxygen-derived free radicals as possible mediators of inflammatory cytokine-induced cell injury, the role of the radicals in the ...

Cardiovascular Research, Original Article, Xiao-Shu Cheng, Hiroaki Shimokawa, Hidetoshi Momii, Jun-ichi Oyama, Naoto Fukuyama, Kensuke Egashira, Hiroe Nakazawa, Akira Takeshita

Date : 01/06/1999
Ehjcardiovascrec

[...]the remarkable role of nitric oxide (NO) as a modulator of biological phenomena has led to the question of its involvement within the spectrum of cardiovascular disease. The resulting research ...

Cardiovascular Research, Review, Roby D Rakhit, Richard J Edwards, Michael S Marber

Date : 15/08/1999
Ehjcardiovascrec

The potential role of, and mechanisms responsible for, beat-to-beat alternations in the amplitude of systolic Ca2+ transients (Ca2+ alternans) was investigated in a canine post-myocardial infarction ...

Cardiovascular Research, Original Article, Andriy E. Belevych, Dmitry Terentyev, Serge Viatchenko-Karpinski, Radmila Terentyeva, Arun Sridhar, Yoshinori Nishijima, Lance D. Wilson, Arturo J. ...

Date : 01/12/2009
Ehjcardiovascrec

Abstract Objective: We tested the hypothesis that simultaneous inhibition of the endothelial integrin αvβ3 and the platelet glycoprotein IIb/IIIa receptor will substantially reduce infarct size ...

Cardiovascular Research, Original Article, Tadamichi Sakuma, Ibrahim Sari, Craig N. Goodman, Jonathan R. Lindner, Alexander L. Klibanov, Sanjiv Kaul

Date : 01/06/2005