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Whether cardiac mitochondria have an innate capacity to temporally self-modulate their membrane potential as a possible adaptive mechanism in the context of cardiac ischemia and early reperfusion is ...

Cardiovascular Research, Review, Michael N. Sack

Date : 01/11/2006 Item size : 474381 bytes

Introduction The structure and biochemical functions of cardiac mitochondria are altered during ischaemiareperfusion (IR).1,2 Impaired mitochondrial function after ischaemia are disastrous for the ...

Cardiovascular Research, ORIGINAL ARTICLES: SPOTLIGHT ON REDUCING MYOCARDIAL ISCHAEMIA/REPERFUSION INJURY, Vu Thi Thu, Hyoung-Kyu Kim, Le Thanh Long, Sung-Ryul Lee, Tran My Hanh, Tae Hee Ko, Hye-Jin ...

Date : 01/05/2012 Item size : 309252 bytes

[...]recent evidence indicates that the contribution of mitochondria to cell injury goes well beyond their role as death executors through the opening of mitochondrial permeability transition pore ...

Cardiovascular Research, EDITORIAL: FOCUS ON MITOCHONDRIA IN CARDIAC DISEASE, Derek J. Hausenloy, Marisol Ruiz-Meana

Date : 01/10/2010 Item size : 156284 bytes

Methods: Inducible nitric oxide synthase (iNOS) expression, cardiomyocyte calcium handling, mitochondrial energetics, and the activation of protective signalling pathways in response to IL-6 were ...

Cardiovascular Research, Original Article, Nicola Smart, Mart H. Mojet, David S. Latchman, Michael S. Marber, Michael R. Duchen, Richard J. Heads

Date : 01/01/2006 Item size : 304116 bytes

Abstract Although the specific roles of nitric oxide (NO) in the heart in general and on cardiac mitochondria in particular remain controversial, it is now clear that both endogenous and exogenous ...

Cardiovascular Research, Review, Sean M. Davidson, Michael R. Duchen

Date : 01/07/2006 Item size : 551284 bytes

Abstract Background: Recent studies have postulated that mitochondrial ATP-sensitive potassium (mitoKATP) channel activation may modulate mitochondrial function with the resultant induction of a ...

Cardiovascular Research, Original Article, Jan Minners, Ewout J van den Bos, Derek M Yellon, Herzl Schwalb, Lionel H Opie, Michael N Sack

Date : 01/07/2000

The MAS is a biochemical system that translocates reducing power from the cytosol into the mitochondria, thus linking glycolysis with the electron transport chain.11 Although previous studies have ...

Cardiovascular Research, EDITORIAL: FOCUS ON MITOCHONDRIA IN CARDIAC DISEASE, Alejandra González-Loyola, Ignasi Barba

Date : 01/11/2010

Methods and results Expression of optic atrophy 1 (OPA1), a mitochondrial fusion protein, was decreased in both human and rat HF, as observed by western blotting. Confocal and electron microscopy ...

Cardiovascular Research, Original Article, Le Chen, Qizhi Gong, James P. Stice, Anne A. Knowlton

Date : 01/10/2009

Consistently, H2O2PC-attenuated I/R-induced cytosolic and mitochondrial Ca2+ overload, Ca2+ transient suppression, mitochondrial reactive oxygen species burst, and loss of mitochondrial inner ...

Cardiovascular Research, Cardiac biology and remodelling, Yixiong Chen, Jinlong Liu, Yanjun Zheng, Jinxi Wang, Zhihua Wang, Shanshan Gu, Jiliang Tan, Qing Jing, Huangtian Yang

Date : 01/02/2015

Several mechanisms, which are not mutually exclusive, have been suggested to be involved in this process2 but formation of a multiprotein complex involving both the inner and the outer membranes, ...

Cardiovascular Research, Cardiac biology and remodelling, Stéphanie Paradis, Valerio Leoni, Claudio Caccia, Alain Berdeaux, Didier Morin

Date : 01/06/2013