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There are increasing data indicating that NO modulates various proteins involved in excitationcontraction coupling (ECC), and here we discuss the evidence that NO may modulate the function of the ...

Cardiovascular Research, Review, Gregory Lim, Luigi Venetucci, David A. Eisner, Barbara Casadei

Date : 15/01/2008 Item size : 401558 bytes

Evidence is now emerging that hyperphosphorylation of the cardiac ryanodine receptor is an important event in chronic heart failure, contributing to impaired contraction and the generation of ...

Cardiovascular Research, Review, Mark Scoote, Alan J Williams

Date : 01/12/2002 Item size : 402108 bytes

Ucn effects were independent of protein kinase A (PKA) activation but were significantly reduced by protein kinase C (PKC) and mitogen-activated protein kinase (MAPK) inhibitors and by brefeldin A, ...

Cardiovascular Research, Original Article, Eva Calderón-Sanchez, Carmen Delgado, Gema Ruiz-Hurtado, Alejandro Domínguez-Rodríguez, Victoria Cachofeiro, María Rodríguez-Moyano, Ana ...

Date : 01/09/2009 Item size : 303590 bytes

Mechanisms involving adaptation of calcium handling during excitationcontraction coupling to late preconditioning remain to be established. [...]we investigated whether the late preconditioned ...

Cardiovascular Research, Original Article, Laurence Lucats, Laurent Vinet, Alain Bizé, Xavier Monnet, Didier Morin, Jin Bo Su, Patricia Rouet-Benzineb, Olivier Cazorla, Jean-Jacques Mercadier, Luc ...

Date : 01/02/2007 Item size : 266637 bytes

Recap on NOS distribution and function in cardiovascular tissues Nitric oxide (NO) is a key universal signalling molecule in the cardiovascular system, acting on both acute and more chronic ...

Cardiovascular Research, REVIEWS: SPOTLIGHT ON REDUCING MYOCARDIAL ISCHAEMIA/REPERFUSION INJURY, Boris Manoury, Virginie Montiel, Jean-Luc Balligand

Date : 01/05/2012 Item size : 553690 bytes

Studies have revealed substantial ultrastructural alterations in several parts of the cardiomyocytes during cardiac disease, including the sarcoplasmic reticulum (SR)2 and transverse (T)-tubules,3,4 ...

Cardiovascular Research, Editorial, Fredrik Swift, Geir Christensen

Date : 01/09/2012 Item size : 179116 bytes

Methods: We used formamide-induced detubulation of mouse ventricular myocytes to investigate differential functional distribution and regulation by PKA of α1 and α2 in T-tubule versus SSL ...

Cardiovascular Research, Original Article, Roger G. Berry, Sanda Despa, William Fuller, Donald M. Bers, Michael J. Shattock

Date : 01/01/2007

Abstract Aims Mutations in the cardiac ryanodine receptor Ca2+ release channel, RyR2, underlie catecholaminergic polymorphic ventricular tachycardia (CPVT), an inherited life-threatening ...

Cardiovascular Research, Original Article, Simon Sedej, Frank R. Heinzel, Stefanie Walther, Nataliya Dybkova, Paulina Wakula, Jan Groborz, Phillip Gronau, Lars S. Maier, Marc A. Vos, F. Anthony Lai, ...

Date : 01/07/2010

In addition to SOCE, there is also growing evidence of another non-voltage-gated Ca2+ entry pathway, store-independent Ca2+ entry (SICE), primarily characterized as an endogenous arachidonic ...

Cardiovascular Research, Editorial, Helen E. Collins, John C. Chatham

Date : 01/03/2015

Introduction The sarcoplasmic reticulum (SR) is a key integrator of excitationcontraction coupling (ECC), transducing the intracellular milieu to synchronize the spatio-temporal dynamics of Ca2+ ...

Cardiovascular Research, Review, Christopher H. George

Date : 15/01/2008