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Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Review, Elizabeth A. Woodcock, Xiao-Jun Du, Melissa E. Reichelt, Robert M. Graham

Date : 01/02/2008 Item size : 499739 bytes
Ehjcardiovascrec

Several studies, including investigations in humans, have clearly shown an important pathophysiological role of this pathway in the cardiovascular system and in disease states such as hypertension, ...

Cardiovascular Research, Editorial, Stephan L.M. Peters, Martin C. Michel

Date : 01/07/2007 Item size : 162404 bytes
Ehjcardiovascrec

Abstract Objective The purpose of the present study was to determine whether increased activation of the RhoA/Rho-kinase (ROCK) pathway occurs in diabetic cardiomyopathy and whether acute inhibition ...

Cardiovascular Research, Original Article, Guorong Lin, Graham P. Craig, Lili Zhang, Violet G. Yuen, Michael Allard, John H. McNeill, Kathleen M. MacLeod

Date : 01/07/2007
Ehjcardiovascrec

Abstract Objective The purpose of the present study was to determine whether increased activation of the RhoA/Rho-kinase (ROCK) pathway occurs in diabetic cardiomyopathy and whether acute inhibition ...

Cardiovascular Research, Lin, Guorong, Craig, Graham P., Zhang, Lili, Yuen, Violet G., Allard, Michael, McNeill, John H., MacLeod, Kathleen M.

Date : 01/07/2007 Item size : 275933 bytes
Ehjcardiovascrec

Several studies, including investigations in humans, have clearly shown an important pathophysiological role of this pathway in the cardiovascular system and in disease states such as hypertension, ...

Cardiovascular Research, Peters, Stephan L.M., Michel, Martin C.

Date : 01/07/2007 Item size : 127140 bytes
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008 Item size : 462733 bytes
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008