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Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Review, Elizabeth A. Woodcock, Xiao-Jun Du, Melissa E. Reichelt, Robert M. Graham

Date : 01/02/2008 Item size : 499739 bytes
Ehjcardiovascrec

Abstract Objective: Although it is generally accepted that endurance training improves cardiac function after myocardial infarction the sub-cellular mechanisms are uncertain. The present study ...

Cardiovascular Research, Original Article, Ulrik Wisløff, Jan P. Loennechen, Susan Currie, Godfrey L. Smith, Øyvind Ellingsen

Date : 01/04/2002 Item size : 371926 bytes
Ehjcardiovascrec

Abstract Objective: Although it is generally accepted that endurance training improves cardiac function after myocardial infarction the sub-cellular mechanisms are uncertain. The present study ...

Cardiovascular Research, Wisløff, Ulrik, Loennechen, Jan P., Currie, Susan, Smith, Godfrey L., Ellingsen, Øyvind

Date : 01/04/2002
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008 Item size : 462733 bytes
Ehjournal

[...]LV remodelling and foetal gene expression was reversed by AVV6.βARKct gene therapy. Introduction Despite recent advances in treatment strategies, heart failure (HF) remains a leading cause ...

European Heart Journal, Raake, Philip W.J., Schlegel, Philipp, Ksienzyk, Jan, Reinkober, Julia, Barthelmes, Jens, Schinkel, Stefanie, Pleger, Sven, Mier, Walter, Haberkorn, Uwe, Koch, Walter J., ...

Date : 14/05/2013 Item size : 309165 bytes
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008