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Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Review, Elizabeth A. Woodcock, Xiao-Jun Du, Melissa E. Reichelt, Robert M. Graham

Date : 01/02/2008 Item size : 499739 bytes
Ehjcardiovascrec

KeywordsCardiomyopathyHeart failureContractile functionRemodelingVentricular functionTime for primary review 33 days. 1 Introduction Heart failure was defined by Braunwald as the pathophysiologic ...

Cardiovascular Research, Review, Stefan Hein, Sawa Kostin, Annette Heling, Yoshi Maeno, Jutta Schaper

Date : 14/01/2000 Item size : 261696 bytes
Ehjcardiovascrec

Nitric oxide (NO) influences many aspects of cardiac function including excitationcontraction coupling, myofilament function, oxygen consumption, substrate metabolism, and cell growth and survival ...

Cardiovascular Research, Shah, Ajay M.

Date : 01/06/2005
Ehjcardiovascrec

Time for primary review 33 days. 1 Introduction Heart failure was defined by Braunwald as the pathophysiologic state of impaired cardiac function rendering the heart unable to maintain an output ...

Cardiovascular Research, Hein, Stefan, Kostin, Sawa, Heling, Annette, Maeno, Yoshi, Schaper, Jutta

Date : 14/01/2000
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008 Item size : 462733 bytes
Ehjcardiovascrec

Time for primary review 33 days. 1 Introduction Heart failure was defined by Braunwald as the pathophysiologic state of impaired cardiac function rendering the heart unable to maintain an output ...

Cardiovascular Research, Hein, Stefan, Kostin, Sawa, Heling, Annette, Maeno, Yoshi, Schaper, Jutta

Date : 14/01/2000
Ehjcardiovascrec

For a detailed consideration of physiological vs. pathological hypertrophy (see Dorn, 2007).], resulting from volume overload-induced cardiomyocyte lengthening, without activation of foetal genes3 ...

Cardiovascular Research, Woodcock, Elizabeth A., Du, Xiao-Jun, Reichelt, Melissa E., Graham, Robert M.

Date : 01/02/2008